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Intrahepatic levels and replicative activity of covalently closed circular hepatitis B virus DNA in chronically infected patients.
Mechanisms of induction and action of interferons.
[Forecasting of efficacy of chronic hepatitis B therapy].
Cloning, eukaryotic expression of human ISG20 and preliminary study on the effect of its anti-HBV.
Evidence for a tumoral immune resistance mechanism based on tryptophan degradation by indoleamine 2,3-dioxygenase.
Prevention of hepatitis B virus infection in vivo by entry inhibitors derived from the large envelope protein.
An Insertion/deletion Polymorphism within RERT-lncRNA Modulates Hepatocellular Carcinoma Risk.
Ribavirin regulates hepatitis C virus replication through enhancing interferon-stimulated genes and interleukin 8.
[Chronic hepatitis B: current therapy]
Mx genes show weaker primary response to virus than other interferon-regulated genes.
Genetic variants of interferon-stimulated genes and IL-28B as host prognostic factors of response to combination treatment for chronic hepatitis C.
Interferon-stimulated genes and their antiviral effector functions.
Ribavirin enhances interferon-stimulated gene transcription by activation of the interferon-stimulated response element.
Mycophenolic acid augments interferon-stimulated gene expression and inhibits hepatitis C Virus infection in vitro and in vivo.
IFIT1 is an antiviral protein that recognizes 5\'-triphosphate RNA.
DICER1 loss and Alu RNA induce age-related macular degeneration via the NLRP3 inflammasome and MyD88.
Mice deficient in STAT1 but not STAT2 or IRF9 develop a lethal CD4+ T-cell-mediated disease following infection with lymphocytic choriomeningitis virus.
Differential impact of interferon regulatory factor 7 in initiation of the type I interferon response in the lymphocytic choriomeningitis virus-infected central nervous system versus the periphery.